Martin B J

Arend O, Remky A, Plange N, Martin B J, Harris A

This study found prolonged retinal blood transit (AVP) in normal pressure glaucoma, but this slowing isn't due to capillary loss or altered peripapillary vessel diameters, suggesting other mechanisms impact ocular circulation.

Arend O, Harris A, Martin B J, Remky A

This study measured optic nerve head blood flow, finding epipapillary velocities were significantly higher than macular velocities and uncorrelated, suggesting distinct microvascular dynamics relevant to glaucoma.

Harris A, Anderson D R, Pillunat L, Joos K, Knighton R W, Kagemann L et al.

Laser Doppler flowmetry non-invasively showed optic nerve head blood flow decreases with hyperoxia and increases with hypercapnia, mirroring brain/retina responses. This tool helps assess physiological optic nerve blood flow changes.

Harris A, Spaeth G L, Sergott R C, Katz L J, Cantor L B, Martin B J

In normal-tension glaucoma, betaxolol improved retrobulbar blood flow by reducing vascular resistance, independently of intraocular pressure, unlike timolol. This suggests a potential vascular benefit for betaxolol in NTG.

Harris A, Sergott R C, Spaeth G L, Katz J L, Shoemaker J A, Martin B J

NTG patients showed higher ophthalmic artery resistance, which improved with CO2, suggesting reversible vasospasm as a potential mechanism for this glaucoma type.

Harris A, Malinovsky V E, Cantor L B, Henderson P A, Martin B J

Studying exercise's effect on eye pressure, researchers found maintaining normal CO2 levels during isometric handgrip exercise prevented the typical IOP drop, suggesting hypocapnia, not exercise itself, lowers IOP.